Abstract
A relationship between the oxygen metabolism of circulating neutrophils and the intensity of lipid peroxidation in exhaled air condensate was studied in chronic bronchitis, ischemic heart disease, and in their combination. An increase in the functional reserve of neutrophils caused by ischemic heart disease promotes the realization of the bio-oxidant potential of these cells in the bronchoalveolar system and intensifies local processes of lipid peroxidation.
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