Abstract

Seizures are a common manifestation of autoimmune encephalitis (AIE), and in some cases may not respond to antiseizure medications. Epileptogenesis in AIE is likely to arise from the synergistic effect of both innately driven neuroinflammation and antibody-induced hyperexcitability. We describe a case of concurrent anti-AMPAR encephalitis and thymomatous myasthenia with refractory focal status epilepticus.

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