Abstract
Accumulated evidence supports the contention that ionizing radiation therapy (RT) effects on tumor-host interactions extend beyond its ability to eliminate the cancer cells within a tumor. The ability of RT to generate inflammatory signals, to enhance the penetration of antigen-presenting cells (APC) and effector T-cells into solid tumors, to free tumor-derived antigens from the dying cancer cells, and to enhance the expression of some surface receptors on the cancer cells that survive, may all be important determinants of the effects of RT on tumor immunogenicity.
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