Abstract
RATIONALE: RRP is characterized by recurrent benign tumors of the respiratory tract, caused by infection with human papillomavirus (HPV). The immunologic mechanism(s) that govern disease variation in RRP, and the factors that predispose only a small group of HPV-exposed individuals to develop RRP, remain unresolved. We have recently reported that RRP is a TH2-like disease, with an imbalance of IL-10 vs. IFNγ, IL-12 and IL-18 expression. We hypothesized that dendritic cells play an important role in establishing this imbalance.
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