Abstract
Results on the mode of recovery of inhibited cholinesterase both in American cockroaches and house flies were found indicative of a predominant recovery process that was not influenced by inhibitors which resulted in dimethyl, di- n-propyl, diisopropyl, and di- n-butyl phosphorylated cholinesterase. A study was made of both in vivo and in vitro ageing of dimethyl phosphorylated cholinesterase. The slow in vivo ageing rate found suggests that a considerable portion of non-aged dimethyl phosphorylated cholinesterase was present even during the prolonged slow recovery of cholinesterase leves. The in vivo ageing experiments combined with the slow in vivo recovery observed in other experiments can best be explained by concluding that the slow recovery is a result of cholinesterase synthesis and not a reversal of inhibited cholinesterase.
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