Abstract
IT has recently been shown that L-3,4-dihydroxyphenylalanine (DOPA) depresses the short-latency reflex paths from the flexor reflex afferents (FRA) and releases long-latency reflex paths from these same afferents, which give a prolonged intense activation of ipsilateral flexor and contralateral extensor motoneurones and a concurrent depolarization of Ia primary afferent fibres1,2. In the normal acute spinal cat the long-latency paths are presumably inhibited by the short-latency paths.
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