Abstract
While the receptor apparatus of the labyrinth reflexes on the skeletal and the ocular muscles has been studied in much detail, there is a lack of such information regarding the labyrinthine reflexes on the vegetative nervous system. Since the latter reflexes form an essential part of the various types of sickness caused by motion, it seemed desirable to study this problem. As a first step, the vasomotor reaction induced by labyrinthine stimulation was chosen, the central and the effector mechanism of this reaction having been analyzed previously (Spiegel and Demetriades 1 ). The starting point of these experiments was a peculiar observation that at first glance seemed in agreement with the theory proposed by Quix 2 that seasickness is caused by stimulation of the otolithic apparatus. In cats in which for other reasons bilateral puncture of the round windows had been performed (Spiegel and Scala 3 ) rotation failed to produce a postrotatory nystagmus. However,
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