Abstract
It is difficult to know where to begin an article on gastritis under the title of 'Recent advances'. Following the discovery of Helicobacter pylori in 1983, I our concepts of gastritis have had to be completely revised. Not only can we now recognize the major cause of 'chronic nonspecific gastritis', but the identification of H. pylori has also allowed us to delineate new forms like reflux 2 and lymphocytic gastritis) Furthermore we can now appreciate that acute 4 and atrophic gastritis s can be part of the spectrum ofH. pylori related inflammation. Given that this entire topic is in a state of flux, it seems reasonable that this article should concentrate on H. pylori gastritis and the two 'special' forms of gastritis, reflux (reactive) and lymphocytic gastritis. I shall examine the main histopathological aspects of these conditions, many of which have only recently been adequately explained.
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