Abstract

Kienböck’s disease is a rare disease described as progressive avascular osteonecrosis of the lunate. The typical manifestations include a unilateral reduction in wrist motion with accompanying pain and swelling. Besides recent advances in treatment options, the etiology and pathophysiology of the disease remain poorly understood. Common risk factors include anatomical features including ulnar variance, differences in blood supply, increased intraosseous pressure along with direct trauma, and environmental influence. The staging of Kienböck’s disease depends mainly on radiographic characteristics assessed according to the modified Lichtman scale. The selection of treatment options is often challenging, as radiographic features may not correspond directly to initial clinical symptoms and differ among age groups. At the earliest stages of Kienböck disease, the nonoperative, unloading management is generally preferred. Patients with negative ulnar variance are usually treated with radial shortening osteotomy. For patients with positive or neutral ulnar variance, a capitate shortening osteotomy is a recommended option. One of the most recent surgical techniques used in Stage III Kienböck cases is vascularized bone grafting. One of the most promising procedures is a vascularized, pedicled, scaphoid graft combined with partial radioscaphoid arthrodesis. This technique provides excellent pain management and prevents carpal collapse. In stage IV, salvage procedures including total wrist fusion or total wrist arthroplasty are often required.

Highlights

  • Kienböck’s disease is described as avascular osteonecrosis of the lunate

  • The anatomical risk factors include the shape of the lunate and distal radius, ulnar variance, the coverage of lunate by radius, the blood supply, the excessive intraosseous pressure, and the venous stasis [2]

  • In line with this study, Daecke et al, with a long- term follow-up study, proved that radial shortening osteotomy along with vascularized pisiform bone grafting is an effective method in treating patients with negative ulnar variance [19]

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Summary

Introduction

Kienböck’s disease is described as avascular osteonecrosis of the lunate. The etiology of this disease remains unknown and controversial [1]. It is possible to determine factors that generally impact the risk of incidence. The anatomical risk factors include the shape of the lunate and distal radius, ulnar variance, the coverage of lunate by radius, the blood supply, the excessive intraosseous pressure, and the venous stasis [2]. Personal factors include age and gender, the associated diseases, the trauma-related factors, the social and environmental factors, and the association with osteonecrosis of other carpal bones [2]. Kienböck’s disease shows male predominance, with a peak incidence in patients aged. The Lichtman scale is the most common radiological classification used to describe Kienböck’s disease. The arthroscopic classification was developed by Bain et al It is based on the number of articular surfaces of the lunate and adjacent articulation, which are defined as non-functional [9]

Staging
Treatment
Stage I (Lichtman Stage I)
Stage II (Lichtman Stage II, Bain 0, Schmitt Stage A)
Stage IIIA (Lichtman Stage IIIA, Bain 1, Schmitt Stage B)
Stage IIIB, IIIC
Procedure
Stage IV
Photographs a patient undergoing vascularized capitate
Summary
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