Abstract
No abstract available
Highlights
Livedoid vasculopathy (LV) is a thrombotic cutaneous disease characterized by episodic painful leg ulceration
Skin biopsy taken from medial side of the ankle on left side showed typical histopathological features of livedoid vasculopathy i.e. mild hyperkeratosis and acanthosis of epidermis, thickening and hyalinisation of dermal vessels with evidence of almost obstructed lumen and fibrinoid material contained in some of the vessel walls
Thereafter, colchicine 0.5 mg twice daily dose was started as an adjuvant therapy, while aspirin was omitted
Summary
Livedoid vasculopathy (LV) is a thrombotic cutaneous disease characterized by episodic painful leg ulceration. Ulcers are typically seen around malleoli, which are often bilateral and often heal, leaving marked atrophic and disfiguring scars. The condition is common among young and middleaged women. The exact pathogenesis of LV is unclear. It is thought to be caused by systemic dysregulation of coagulation, resulting in the formation of fibrin thrombi of the superficial dermal plexus. The main goals of the treatment are pain relief, prevention of recurrent ulceration and progressive scarring. Most treatment regimens are based on anecdotal off-label protocols. Long term remission has always been a challenge
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