Abstract
The authors of this study note that postmortem markers of Alzheimer disease and other dementias may be distinct from the self-report measure of cognitive activity, which they state provides no support for the reverse-causality hypothesis.1 The authors suggest that cognitive activities may lead to changes in brain structure and function that could enhance cognitive reserve. Is it not just as likely that there are features of brain structure or function that have not been discerned in postmortem evaluation, thus failing to fully disprove the reverse-causality hypothesis?
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