Abstract
Periodontitis is one of the most prevalent oral diseases. The study of its pathogenesis has advanced greatly in recent years. However, the details of the mechanisms involved in bone resorption are not clear yet. RANK, RANKL, and OPG areproteins that have a known role in other bone tissue diseases. This paper reviews the evidence on their potential participation in the pathogenesis of periodontal disease. More evidence is needed to understand the relationship between RANK, RANKL, OPG, and bone resorption in periodontitis.
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