Abstract
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Highlights
I have had a long term interest in the cause of pyloric stenosis (PS) of Infancy and read this paper with interest (Infantile hypertrophic pyloric stenosis following repair of H-type tracheoesophageal fistula repair and post-operative erythromycin therapy; published in 1st issue of volume 8, 2019).[1]
I believe an inherited larger than normal Parietal cell mass explains all the clinical features.[2]
Primary hyperacidity may be considered as an explanation for the well-known increase in the incidence of PS in babies with TOF and EA
Summary
I have had a long term interest in the cause of pyloric stenosis (PS) of Infancy and read this paper with interest (Infantile hypertrophic pyloric stenosis following repair of H-type tracheoesophageal fistula repair and post-operative erythromycin therapy; published in 1st issue of volume 8, 2019).[1].
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