Abstract
Copy number variants (CNVs) have a fundamental role in human disease and population diversity (1-3). Exploration of rare CNVs with genomic microarrays has allowed rapid assessment of structural variation at high resolution and low cost. Studies have implicated various schizophrenia associated CNVs, including deletions on 22q11, 15q13, 1q21.1, 2p16.3, and 15q11.2 and the duplications of 16p11.2. CNVs vary in frequency and with larger sample sizes and greater power. CNVs that are more rare and with less effect are continually being updated.
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