Abstract

A 56 years old female, body mass index 44 kg/m 2 , postmenopausal patient who had a solid mass of approximately 6-cm in diameter at the left adnexal area underwent laparotomy with vertical incision. Staging surgery was performed because of the frozen section result that was reported as a malignant ovarian cancer. On the postoperative 2 nd day, the patient had dyspnea, cold sweats, tachycardia, tachypnea and bowel sounds were detected as hypoactive. SO 2 was measured as 88% during follow-up and right costophrenic angle was appearing blunt in her chest X-ray. Arterial blood gas analysis results were as follows; ph: 7.41, PCO 2 : 29.2 mmHg, SO 2 : 86%, HCO 3 : 18.2 mmol/L, and base excess (BE): -4.9 mmol/L. There was no additional feature in the blood cell counts and biochemical tests. The diagnosis of pulmonary emboli was ruled out with thoracic spiral tomography. In the sequel, the patient began vomiting, while her bowel sounds were remained hypoactive with addition of abdominal distension. After the initial intravesical pressure (IVP) was measured as 16 mmHg (22 cmH 2 O) manually, the patient was observed with endoluminal decompression methods and medical management. The clinical signs and physical examination findings did not improve; IVP measured two hours later was 21 mmHg (28 cmH 2 O). Because of these clinical findings and the rise of sequential IVP measurements, the patient was diagnosed with abdominal compartment syndrome. Decompressive laparotomy was performed and completed by closing the skin without suturing the fascia. Eight months later, her overall condition was good and follow-up has continued. Monitoring intraabdominal pressure with intermittent indirect IVP measurements in intensive care patients with high risk for ACS has great significance for early diagnosis, increasing the awareness for this condition. Early decompressive surgery in ACS is the life-saving step.

Highlights

  • Abdominal compartment syndrome (ACS) is a progressive clinical condition characterized by the persistent increase in intraabdominal pressure (IAP) that may result in sepsis and even multiple organ failure

  • The World Society of the Abdominal Compartment Syndrome (WSACS) defines intraabdominal hypertension (IAH) as sustained or repeated pathologic elevation of IAP ≥12 mmHg, and ACS as a sustained IAP >20 mmHg that is associated with new organ dysfunction (2)

  • ACS was managed with Decompressive laparotomy (DL) which was terminated by closing the skin and leaving the fascia open that was defined as planned incisional hernia in literature

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Summary

INTRODUCTION

Abdominal compartment syndrome (ACS) is a progressive clinical condition characterized by the persistent increase in intraabdominal pressure (IAP) that may result in sepsis and even multiple organ failure. The patient began vomiting, while her bowel sounds remained hypoactive with the addition of abdominal distension Her oral intake was stopped, and a urinary catheter was placed (>100cc/hr). There was no change in control arterial blood gas and laboratory analysis; the patient’s dyspnea, tachypnea, tachycardia, and vomiting continued, and physical examination findings did not improve; IVP that measured two hours later was 21 mmHg (28 cmH2O). There was no bacterial growth in the culture of intraabdominal fluid All of her abnormal signs improved on the 5th day and enteral feeding was initiated. After three months from completion of the chemotherapy, the loop colostomy was closed Her overall condition was good, and follow-up has continued

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