Abstract
The aorta of hypertensive rabbits has an increased content of water, sodium and fibrinogen. Fibrinogen catabolism is normal in most hypertensive animals and is, therefore, not a concomitant of increased vascular permeability but accelerated catabolism can be demonstrated when there is associated shock, red cell fragmentation due to DOCA angiopathy, hypertension in pregnancy, coincidental anoxia and acidosis, or in terminal uraemia. Under all these conditions, fibrin is deposited particularly in the kidney and may, therefore lead to further renal ischaemia.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
