Abstract
Autoantibodies (ABs), often deemed pathological, have received increasing attention in the last 2 decades. Nevertheless, admittedly, we still do not know much about their physiological role nor about the circumstances that may turn them into pathologically relevant agents. Nagele et al. (1) reported early on that natural ABs of the IgG class are abundant and ubiquitous in human sera, and their number is influenced by age, gender, and disease. Even earlier, Cohen (2) postulated that human ABs of all classes are organized in clusters and that cluster analyses can potentially help differentiate healthy from disease states, demanding the development of new tools to undertake large-scale AB analyses (2).
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