Abstract
Fast-food consumption is highly prevalent in the United States (U.S.) and associated with greater risk of diabetes and cardiovascular disease.1 However, little is known about the impact of fast-food consumption on the risk of nonalcoholic fatty liver disease (NAFLD), which affects over 30% of the U.S. population, or whether the effect of fast food on steatosis is different among persons with metabolic risk factors, including obesity and diabetes. Dietary intake has been hypothesized to be one of the most influential modifiable factors for NAFLD, yet there are major gaps in evidence linking diet with steatosis, particularly among high-risk groups, limiting public health efforts around dietary prevention and risk mitigation strategies to reduce fatty liver.
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