Abstract
The basic building block of cell nuclei is postulated to be a macromolecule consisting of an exon of DNA linked laterally with a polypeptide whose DNA-distalmost NH 2 radical is in an electronic pulsatory state which constitutes a centriole. The pulsating centriole impresses oscillating excitation along the polypeptide to the DNA rendering the DNA partially oxidative of substances in the fluid environment. This oxidation activates a second type of oscillating excitation distad along the polypeptide, keeping the centriole in its pulsatory state. The exon-polypeptide-centriole macromolecule is, in brief, in unique inter-end-dependent steadystate dynamic equilibrium. Replication of the exon and then the polypeptide occurs through oscillatory resonance-attraction, seriatim, of identical sub-entities from the fluid environment. Final duplication of the centriole occurs through pulsatory resonance-induction acting upon the daughter polypeptide's terminal NH 2. The resultant daughter centriole is, however, in 180° opposite pulsatory phase. Opposite-phase pulsators repel one-another; like-phase pulsators attract one-another. These postulated pulsator, and oscillator, principles afford explanation of the main phenomena of mitosis. Pathological foreshortening in polypeptide replication offers explanation of malignant mitosis.
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