Abstract

Cardiac manifestations in hyperthyroidism (HT) are very common and have varied clinical aspects. Reports of tricuspid regurgitation (TR) and pulmonary arterial hypertension (PAH) are, however, infrequent because neither is often symptomatic. TR in HT is functional and secondary to the right ventricle (RV) dilatation as a result of the increased venous return. The exact mechanism of the PAH in HT is not known but could be related to the hyperkinetic state of thyrotoxicosis or an autoimmune phenomenon. Four cases of PAH and TR occurring in association with HT are described.

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