Abstract
Atopic dermatitis (AD) is classified into the IgE-high extrinsic and IgE-normal intrinsic types. While extrinsic AD is the classical type with high prevalence, intrinsic AD occupies approximately 20% of total AD with female predominance [1]. The skin barrier is perturbed in the extrinsic, but not intrinsic type [1], and thus filaggrin gene mutations are not a feature of intrinsic AD [2]. While the two types have comparable frequencies of circulating Th2 cells, intrinsic AD shows a significantly increased [...]
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