Abstract

Electrophoretic and densitometric data revealed induction of apoptosis in the kidney due to experimentally induced ischemia-reperfusion injury. Such apoptosis in the kidney was reduced in rats given 62.5 or 125 mg/kg body wt./day Wen-Pi-Tang orally for 30 days prior to ischemia-reperfusion. An increase in the dose of Wen-Pi-Tang was associated with suppressed fragmentation of DNA, a ladder pattern of low-molecular-weight molecules, resulting in a beneficial effect on renal function.

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