Abstract
There is limited evidence that aneuploidy disproportionately effects outcomes in patients with RPL as compared to the general infertility population. In an effort to investigate the potential that patients with RPL without balanced translocations have a biologic mechanism related to aneuploidy behind their poor pregnancy outcomes, we sought to evaluate if the proportion of aneuploidy within a cohort of embryos impacts pregnancy outcomes. If an intrinsic biologic deficit exists, we suspect that patients with high aneuploidyrates in the RPL population would be disproportionately helped by CCS.
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