Abstract
The exact amount of LH that is required for optimal follicular and oocyte maturation in women undergoing assisted reproduction technology cycles has been the object of debate. A recent study (1) suggested that the mechanism underlying the detrimental effect of low endogenous LH levels could be explained by reduction of the intrafollicular androgen substrate for E2 synthesis, which is suboptimal for oocyte maturation during the follicular phase. However, further studies are needed for a more accurate explanation of the deleterious follicular environment.
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