Abstract
The pancreatic Islet is composed of hundreds of highly heterogeneous, insulin-secreting beta cells. Gap-junction protein channels form links between this complex network of dynamic cells, allowing them to synchronize and secrete insulin in a pulsatile fashion. This synchronization is essential for healthy glucose homeostasis and is lost preceding Type I and Type II Diabetes. Therefore, understanding the beta-cell network and how it reacts to perturbations holds significant promise to new diabetes diagnostics and therapies.
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