Abstract
Because of its relative accessibility, the pial vasculature has often been used as an in vivo model of cerebral vascular smooth muscle control. However, the role of this portion of the cerebral vascular bed in various regulatory phenomena is difficult to assess because of lack of information about the resistance of proximal and distal vascular segments. Measurements of blood pressures in pial arteries and arterioles in the cat (1, 4) suggest that the resistance of the pial vascular bed is only 15 to 20% of total cerebral vascular resistance under normotensive, normocapnic conditions and that the major site of resistance (40 to 60%) is in the parenchymal vessels (arterioles, capillaries, venules).
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