Abstract
Background. We have demonstrated an increased risk of acute gastroenteritis (AG) and community-acquired pneumonia (CAP) in gastroesophageal reflux disease (GERD)-affected children (Berni Canani R et al. Pediatrics, 2006) treated with gastric acidity (GA) inhibitors. These effects could be at least, in part, attributable to modifications of intestinal microflora profile. We aimed to test the hypothesis that probiotics could limit the risk of infection deriving from GA-inhibitor therapy by positive influence on intestinal microflora composition.
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