Abstract
The objective of this study was to assess the prevalence of mucositis and peri-implantitis associated with the use of two types of implants-conventional versus platform switching after one year of loading. A longitudinal study of 64 implants in 25 patients was performed. Clinical variables, such as clinical pocket depth and bleeding upon probing, plaque, mobility, gingival recession, clinical attachment loss, and radiographic bone loss, were analyzed. The case definition for peri-implantitis was established as pockets of ≥ 5 mm with bleeding and bone loss ≥ 2 mm. One year after implant loading, the prevalence of mucositis and peri-implantitis with conventional implants (CIs) was 81.2% and 15.6%, respectively. For platform switching implants (PSIs) the prevalence was 90% and 6.6%, respectively. These differences were not statistically significant (p = 0.5375). However, there was a trend towards a lower prevalence of peri-implantitis with platform switching Implants.
Highlights
The functional and aesthetic restoration of partially or totally edentulous areas with dental implants is widely accepted
We evaluated the prevalence of mucositis and peri-implantitis in two different types of implants (PSI vs. conventional implant system (CIs)) after one year of loading
A cross-sectional study was conducted to assess the prevalence of mucositis and peri-implantitis
Summary
The functional and aesthetic restoration of partially or totally edentulous areas with dental implants is widely accepted. Survival rates for dental implants evaluated in several systematic reviews and meta-analyses are > 90% and depend on the time of evaluation. Modifications to conventional implant system (CIs) have been proposed in search of better restorative, aesthetic, and biological results. Platform switching implants system (PSIs) utilize an abutment with a smaller diameter aim to preserve the crestal bone level.[3,4] Most studies have compared the radiographic bone level between PSIs and CIs. Some studies have proposed that crestal bone stability is a consequence of the internal displacement of the abutment-implant junction, which displaces the inflammatory infiltrate away from the crestal bone and creates a space for the formation of biologic width.[3,4,5,6]
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