Abstract
Abstract Low dosages of chlorine rapidly produced high levels of NPOX, but very little POX was formed. Chlorine dioxide produced over 80 percent less TOX than chlorine. TOX formation at low chlorine dosages was a function of the chlorine dose whereas, in the presence of excess chlorine it vas a function of the DOC. Low dosages of chlorine dioxide and ozone had little effect on TOX precursors directly and did not enhance GAC adsorption of TOX precursors. Use of high dosages of chlorine dioxide or ozone prior to GAC increased the DOC adsorption capacity of the carbon.
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