Abstract
Maternal smoking is a major risk factor for SIDS. The mechanism of action may be a nicotine mediated attenuation of the hypoxic defense as evidenced by impaired arousal and blunted ventilatory response to acute hypoxemia in young lambs exposed to an acute nicotine infusion (Hafstrom et al, Pediatr Res 39:386A, 1996). The aim of this study was to determine whether prenatal nicotine exposure alters the cardiorespiratory response to hypoxia after birth.
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