Abstract
Background and aims In recent years, evidence has shown that prenatal trafficrelated air pollution exposure influences fetal growth. Changes in mitochondrial DNA (mtDNA) content may represent a biologically relevant endpoint on the mechanisms underlying the association between air pollution and fetal growth restrictions. In this study, we aimed to assess the role of placental mtDNA content on the association of prenatal NO2 exposure with fetal growth assessed by ultrasound measurements.
Highlights
Background and aimsIn recent years, evidence has shown that prenatal trafficrelated air pollution exposure influences fetal growth
biologically relevant endpoint on the mechanisms underlying the association between air pollution
we aimed to assess the role of placental mitochondrial DNA (mtDNA) content on the association
Summary
Evidence has shown that prenatal trafficrelated air pollution exposure influences fetal growth. Changes in mitochondrial DNA (mtDNA) content may represent a biologically relevant endpoint on the mechanisms underlying the association between air pollution and fetal growth restrictions. We aimed to assess the role of placental mtDNA content on the association of prenatal NO2 exposure with fetal growth assessed by ultrasound measurements
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