Abstract

The association of persistent tachyarrhythmias, “idiopathic” cardiomyopathy (ICM), and heart failure (HF) was first described about a century ago. Subsequent observations of the reversibility of ICM and HF after successful treatment of tachyarrhythmias provided the proof of concept that ICM and HF could be consequences of tachycardia, contrary to the traditional teaching that ICM and HF were causative to tachyarrhythmias due to preexisting pathophysiological substrates. Similarly, premature ventricular contractions (PVCs) were viewed as markers of underlying cardiac dysfunction rather than causes of cardiac dysfunction.

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