Abstract

BackgroundWe evaluated the effect of transvaginal mesh (TVM) surgery for voiding function and continence using noninvasive examination and questionnaire. The present study aimed to ascertain which categories of patients need concomitant mid-urethral sling (MUS) after TVM surgery.MethodsWe included women who underwent TVM procedure between November 2009 and October 2013. Data from noninstrumented uroflowmetry and questionnaires about urinary symptoms were analyzed.ResultsThe present study investigated the cases of 961 women who underwent TVM surgery. The persistence of stress urinary incontinence (SUI) was 57.6%. Almost all the parameters measured using uroflowmetry and questionnaires significantly improved in all types of urinary incontinence 12 months after surgery. A history of hysterectomy, preoperative high flow (corrected maximum flow rate > 1.5), and preoperative urge urinary incontinence were independent risk factors for the persistence of SUI.ConclusionsTVM for pelvic organ prolapse improved subjective and objective voiding function. Mixed urinary incontinence (MUI) patients with high urinary flow may be suitable for concomitant MUS with TVM because of the high level of SUI persistence.

Highlights

  • We evaluated the effect of transvaginal mesh (TVM) surgery on voiding function and continence using noninvasive examination and questionnaire

  • There was no significant difference between the urge urinary incontinence (UUI) persistence in preoperative Mixed urinary incontinence (MUI)

  • This study identified a history of hysterectomy, preoperative high flow, and preoperative UUI as independent risk factors for stress urinary incontinence (SUI) persistence (p < 0.05)

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Summary

Introduction

We evaluated the effect of transvaginal mesh (TVM) surgery for voiding function and continence using noninvasive examination and questionnaire. The present study aimed to ascertain which categories of patients need concomitant mid-urethral sling (MUS) after TVM surgery. Pelvic organ prolapse (POP) is frequent, with a prevalence that ranges from 2.9 to 11.4% when assessed by questionnaire and from 31.8 to 97.7% when evaluated by clinical examination [1]. Voiding dysfunction may be attributed to bladder outlet obstruction because of mechanical urethral kinking, or urethral and/or bladder neck compression by the prolapsed cystocele [3]. Uterine prolapse or rectocele may contribute to bladder outlet obstruction. Overactive bladder and POP are strongly correlated, and bladder outlet obstruction is likely the cause of this relationship [4]

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