Abstract
Some of the views on rapid-eye-movement sleep behaviour disorder (RBD) have changed greatly during the 25 years since the disease was first characterised in human beings.1 Initially RBD was regarded as an intriguing and supposedly rare parasomnia. This notion has expanded into the appreciation that RBD might be an early clinical manifestation of an evolving neurodegenerative disorder.2–9 Ample evidence now exists to show that RBD is often associated with neurodegenerative disorders, particularly the synucleinopathies—Parkinson's disease, dementia with Lewy bodies, multiple system atrophy, and pure autonomic failure.
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