Abstract

An increased risk for hemorrhagic fever with renal syndrome caused by Puumala hantavirus was forecast for Sweden in 2007. The forecast was based on a predicted increase in the number of Myodes glareolus rodents (reservoir hosts). Despite raised awareness and preparedness, the number of human cases during July 2007–June 2008 was 1,483, a new high.

Highlights

  • An increased risk for hemorrhagic fever with renal syndrome caused by Puumala hantavirus was forecast for Sweden in 2007

  • Puumala virus (PUUV) is excreted from bank voles by saliva, urine, and feces, and transmission to humans often occurs by inhalation of aerosolized excreta

  • An average of 10–40 hemorrhagic fever with renal syndrome (HFRS) cases are serologically confirmed per 100,000 population every calendar year in HFRS-endemic northern Sweden [5,6]

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Summary

Predicting High Risk for Human

An increased risk for hemorrhagic fever with renal syndrome caused by Puumala hantavirus was forecast for Sweden in 2007. Puumala virus (PUUV) is the etiologic agent of nephropathia epidemica, a mild form of hemorrhagic fever with renal syndrome (HFRS). PUUV is likely to be the most prevalent hantavirus in Europe [1]; the bank vole Myodes (Clethrionomys) glareolus, one of the most widespread and abundant mammal species on the continent, is a natural reservoir [2,3,4]. A total of 807 cases, or 313 cases/100,000 population, were reported from Västerbotten County, a HFRS-endemic area.

The Study HFRS has been a reportable disease in Sweden since
No HFRS cases
Conclusions
Findings
Snow cover data were collected and kindly provided by Mats

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