Abstract
Local production of estrogen via upregulated aromatase (CYP19, p450 arom) expression contributes to the persistence and self-propagation of endometriotic lesions. PPAR-γ ligands have been shown to decrease p450 arom in human granulosa cells. We sought to determine if a PPAR-γ ligand, ciglitazone (CIG), could affect aromatase expression in endometrial stromal cells.
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