Abstract
uptured intracranial aneurysms are not an uncommon cause of severe neurologic damage and death. RecurR rent rupture and vasospasm are, by far, the leading causes of worsening prognosis for the survivors. Delayed ischemic neurologic deficit (DIND), caused by narrowing of cerebral vessels following subarachnoid hemorrhage (SAH), continues to burden patients as a major cause of morbidity and mortality. Angiographic vasospasm occurs in 30% 70% of the cases after SAH, rendering 30% of the patients with symptomatic DIND. Symptoms develop gradually and may fluctuate. In the awake patient, vasospasm development may include new or increasing headache, declining of consciousness, and, depending on the anatomic regions of decreased perfusion, hemiparesis, aphasia, abulia, and other focal signs.
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