Abstract
Potassium chloride applied topically to the neocortex facilitates lordosis behavior in estrogen-primed ovariectomized rats. This effect is absent in rats that are also adrenalectomized. Potassium chloride fails to facilitate lordosis in estrogen-primed rats, with intact adrenals, which are pretreated with dexamethasone, an inhibitor of the release of adrenocorticotropic hormone. Adrenalectomized rats that have been treated with dexamethasone are capable of responding to progesterone with the display of lordosis. The results suggest that application of potassium chloride to the neocortex acts as a stressor that causes the release of adrenal progestin.
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