Abstract
Potassium chloride applied topically to the neocortex facilitates lordosis behavior in estrogen-primed ovariectomized rats. This effect is absent in rats that are also adrenalectomized. Potassium chloride fails to facilitate lordosis in estrogen-primed rats, with intact adrenals, which are pretreated with dexamethasone, an inhibitor of the release of adrenocorticotropic hormone. Adrenalectomized rats that have been treated with dexamethasone are capable of responding to progesterone with the display of lordosis. The results suggest that application of potassium chloride to the neocortex acts as a stressor that causes the release of adrenal progestin.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
