Abstract
Postural orthostatic tachycardia syndrome (POTS) is a heterogeneous disorder of the autonomic nervous system that is defined by symptoms of orthostatic intolerance. According to the current criteria for adults, currently, POTS is defined as a heart rate increment of 30 beats/minute or more after 10 minutes of standing in the absence of orthostatic hypotension. There is a vast majority that remains misdiagnosed due to the heterogeneity of the disorder. Due to a lack of Food and Drug Administration (FDA) approved therapy, alternative therapies and over the counter medications are used to alleviate the symptoms. This is an uncommon presentation observed primarily in women, as it is more prevalent in females.
Highlights
BackgroundPostural orthostatic tachycardia syndrome (POTS) is a disorder of the autonomic nervous system that is defined by symptoms of orthostatic intolerance
Postural orthostatic tachycardia syndrome (POTS) is a heterogeneous disorder of the autonomic nervous system that is defined by symptoms of orthostatic intolerance
Postural orthostatic tachycardia syndrome (POTS) remains to be poorly understood, and many patients suffer without being diagnosed for an extended period
Summary
Postural orthostatic tachycardia syndrome (POTS) is a disorder of the autonomic nervous system that is defined by symptoms of orthostatic intolerance. It is a final, common pathway of a heterogeneous group of underlying diseases that display similar clinical characteristics and are associated with poor health-related quality of life due to debilitating fatigue, poor sleep, and exercise intolerance [1,2,3]. It was seen that a majority of patients with postural orthostatic tachycardia syndrome (POTS) fulfilled criteria for chronic fatigue syndrome (CFS). As we know from the underlying pathophysiology of postural orthostatic tachycardia syndrome (POTS), that it is an autonomic dysfunction, it would likely affect the lower urinary tract functions as well. This leads to unrealistic expectations about the beneficial effects of treatment aimed to correct the postural tachycardia and to secondary frustration and symptom amplification.This indicates that further research on the unusual presentation of patients with postural orthostatic tachycardia syndrome is required
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