Abstract
Increasing evidence suggests that the post-prandial state is a contributing factor to the development of atherosclerosis. In diabetes, the post-prandial phase is characterized by a rapid and large increase in blood glucose levels. The possibility that the post-prandial “hyperglycemic spikes” may be relevant to the onset of cardiovascular complications recently received much attention. Epidemiological studies and preliminary intervention studies have shown that post-prandial hyperglycemia is a direct and independent risk factor for cardiovascular disease. Most of the cardiovascular risk factors are modified in the post-prandial phase in diabetic subjects and directly affected by an acute increase of glycemia. The mechanisms through which acute hyperglycemia exerts its effects may be identified in the
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