Abstract
Background and Aims : Lipoprotein(a) [Lp(a)] is a low-density lipoprotein (LDL)-like particle containing apolipoprotein(a) [apo(a)] that increases risk of atherosclerotic cardiovascular disease (ASCVD) in familial hypercholesterolaemia (FH). Postprandial redistribution of apo(a) protein from Lp(a) to triglyceride-rich lipoproteins (TRL) may also increase the atherogenecity of TRL particles. Omega-3 fatty acid (ω-3FA) supplementation improves postprandial TRL metabolism in FH subjects. However, its effect on postprandial apo(a) metabolism has not yet to be investigated.
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