Abstract
The present series demonstrates that hemodynamic abnormalities persist following intracardiac repair of tetralogy of Fallot despite clinical improvement. The incidence of residual ventricular communication and residual outflow obstruction is consistent with previous reports. The pulmonary incompetence is well tolerated when it exists as an isolated residual abnormality. The use of Teflon cloth to relieve the outflow obstruction has not led to aneurysm formation. This report calls attention to the occurrence of tricuspid insufficiency in patients with tetralogy of Fallot and the increased postoperative morbidity with this combination. Finally, aortic regurgitation is part of the pathophysiology of tetralogy of Fallot and may well affect the results of the intracardiac repair. The long-term consequences of these abnormalities are left to future consideration.
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