Abstract
Posterior Scleritis (PS) is an inflammation of the posterior scleral segment, of low inflammatory or infectious etiology, with a rapid, progressive and irreversible evolution with severe visual impairment, especially if the diagnosis and treatment of (PS) are not performed in time skillful. Ocular pain, eye movement pain, headache and visual haze are the main signs and symptoms. It can be of idiopathic cause or associated with systemic disease in up to 45% of the cases. Rheumatoid arthritis is described as [...]
Highlights
IntroductionPosterior scleritis (PS) is an inflammation of the posterior scleral segment of inflammatory or infectious etiology which is poorly diagnosed and frequently treated late, and can compromise vision and lead to blindness.[1]
Posterior scleritis (PS) is an inflammation of the posterior scleral segment of inflammatory or infectious etiology which is poorly diagnosed and frequently treated late, and can compromise vision and lead to blindness.[1]Ocular pain, eye movement pain, headache, and blurry vision are the main signs and symptoms
Anterior Scleritis (AS) is present in 43% of cases manifesting as hyperemia, photophobia, and tearing, being an important nosological symptom
Summary
Posterior scleritis (PS) is an inflammation of the posterior scleral segment of inflammatory or infectious etiology which is poorly diagnosed and frequently treated late, and can compromise vision and lead to blindness.[1]. Anterior Scleritis (AS) is present in 43% of cases manifesting as hyperemia, photophobia, and tearing, being an important nosological symptom. It has a rapid, progressive and irreversible progression with severe visual impairment, especially if PS is not diagnosed and treated in a timely manner, causing catastrophic damage to noble eye structures. Our objective is to describe a case of a 59-year-old female patient with posterior scleritis without systemic disease associated. IAR, female, 59 years old, Caucasian, started ocular pain in the right eye (RE) 1 month ago, and was examined in two different ophthalmological services by specialists, without precise diagnosis and being treated as chronic conjunctivitis and anterior uveitis. The visual acuity was of LogMar 0.4 with treatment of the initial inflammatory process, both in the posterior pole and papilledema, with some posterior pole pigment alterations persisting (Figures 5, 6 and 7)
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