Abstract
Posterior reversible encephalopathy syndrome (PRES), a term coined in 1996, describes a clinico-radiologic entity characterised by headache, seizures, decreased vision, impaired consciousness and white matter oedema in bilateral occipito-parietal regions [1]. Hypertensive encephalopathy, eclampsia, immunosuppressive/cytotoxic drugs, organ transplantation, renal disease, autoimmune diseases and vasculitides are reported risk factors of PRES [1, 2]. Although potentially reversible, delay in treatment can result in permanent brain damage [3, 4].
Highlights
Posterior reversible encephalopathy syndrome (PRES), a term coined in 1996, describes a clinico-radiologic entity characterised by headache, seizures, decreased vision, impaired consciousness and white matter oedema in bilateral occipito-parietal regions [1]
Our patient presented with PRES as the first manifestation of chronic kidney disease
An acute elevation of blood pressure and the underlying kidney disease were the probable precipitants for her presentation
Summary
Posterior reversible encephalopathy syndrome (PRES), a term coined in 1996, describes a clinico-radiologic entity characterised by headache, seizures, decreased vision, impaired consciousness and white matter oedema in bilateral occipito-parietal regions [1]. Hypertensive encephalopathy, eclampsia, immunosuppressive/cytotoxic drugs, organ transplantation, renal disease, autoimmune diseases and vasculitides are reported risk factors of PRES [1, 2]. Delay in treatment can result in permanent brain damage [3, 4]
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