Abstract
The objective of our study was to describe PET performed in term AGA infants at 36 mo who were diagnosed to have perinatal asphyxia defined as cord ph <7.1, 5 min Apgar ≤4, need for CPR at birth and presence of hypoxic ischemic encephalopathy. Surviving infants had serial neurological examinations categorized at 36 mo as normal, neuromotor impairment with no developmental delay, or cerebral palsy (CP with developmental delay). Infants had neonatal CT and magnetic resonance imaging (MRI) at 6 mo. At 36 mo infants underwent PET to assess glucose metabolism using Siemens EXACT-HR tomograph with IV administration of 0.143 mCi/kg of 18-fluoro-2-deoxyglucose (FDG). PET and neurologic exams were performed by independent examiners.
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