Abstract

The host immune system is not so active against Aspergillum sp. due to the anatomical location of maxillary antrum that facilitates the fungal multiplication. At least 10% of chronic sinusitis are actually infections with Aspergillum, mainly developing the clinical feature of maxillary aspergilloma. Though the terminal respiratory airways are the main access portal of the mycotic infection, the oro-antral fistulas, dental extractions, iatrogenic root perforations which also involve the mucousa of the alveolar ridge might be additional risk factors. It seems that the root canal overfillings may develop a maxillary sinus aspergillosis since zinc from the in sinusal cavity extruded sealers plays the role of a growth factor for Aspergillus. Accordingly, a chronic sinusitis with purulent secretions, refractory to conventional treatment and associated with a foreign body identified on radiographs as root filling material might be suspected as maxillary sinus aspergilossis.

Highlights

  • The host immune system is not so active against Aspergillum sp. due to the anatomical location of maxillary antrum that facilitates the fungal multiplication

  • At least 10% of chronic sinusitis are infections with Aspergillum, mainly developing the clinical feature of maxillary aspergilloma

  • Se pare că aspergiloza sinusurilor maxilare cauzată de obturaţiile de canal cu depăşire este favorizată de prezenţa zincului în cimenturile de sigilare, unul dintre metalele grele care constituie factori de creştere pentru ciupercile din genul Aspergillus [3,18,24,25,26]

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Summary

Introduction

The host immune system is not so active against Aspergillum sp. due to the anatomical location of maxillary antrum that facilitates the fungal multiplication. Agentul patogen cel mai frecvent întâlnit la om în aspergiloza invazivă este specia A. fumigatus (80-90%), secondată de A. flavus (5-10%) şi rareori de A. niger (1-5%), A. terreus (1%) sau A. nidulans [3,4].

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