Abstract
Enteric hyperoxaluria (EH) is caused by excess absorption of dietary oxalate leading to elevated urinary oxalate (UrOx) levels. Once absorbed, oxalate can complex with calcium to form insoluble crystals, and as a result chronically elevated UrOx levels are a major risk factor for the development of kidney stones and progression to kidney damage. There are currently no approved therapies for EH; the standard of care options is limited to supportive measures and dietary restrictions that have relatively low compliance.
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