Abstract
Currently, very few effective strategy is available for the treatment of renal fibrosis induced by renal ischemia reperfusion injury (IRI). M2 macrophages are one of the core regulators in the progression of renal fibrosis. Here, we established a self-assembled glycol-peptide, BIVA-PK, which specifically induced apoptosis of M2 macrophages in the renal interstitium and ameliorated renal fibrosis by modulating the local immune microenvironment.
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