Abstract
Better health care has resulted in increased survival of sickle cell disease (SCD) patients. Consequently, renal abnormalities are becoming a significant cause of morbidity and mortality. SCD individuals have supranormal proximal tubular function, with increased tubular secretion of creatinine, thus creatinine tends to overestimate glomerular filtration rate (GFR). Creatinine based eGFR is believed to be a late marker of nephropathy among these individuals. As such, serum cystatin C is suggested to assess renal function more accurately.
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