Abstract
Fibroblast growth factor 23 (FGF-23), primarily produced by osteocytes, is a phosphate-regulating hormone. The levels of FGF-23 increase along with the decline in kidney function. This occurs as a physiologic response to maintain normal serum phosphate levels and help maintain the average phosphate balance. Dysregulation in iron balance and inflammation occurred in chronic kidney disease, including patients with hemodialysis. This study aimed to determine the difference in serum calcium phosphate levels and iron status between regular hemodialysis patients with high and lower FGF-23 serum levels.
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